Clomiphene citrate – is a synthetic estrogen or antiestrogen drug that has nothing to do with anabolic steroids and capable of selectively providing blocking effect on estrogen.
The main objective of Clomiphene Citrate is a confrontation reverse the effects of estrogen on the body, or rather a chain of HPTA – the hypothalamus – pituitary – testes, increasing the synthesis of LH – luteinizing hormone and GF – follicle-stimulating hormone. This ability is especially appreciated by the men because their bodies gonadotropins LH and FG are involved in the production of testosterone. The drug is important for athletes who use anabolic and androgenic steroids, causing decrease in the level of testosterone. In such cases, it is used to complete the course as an obligatory element of post-cycle therapy.
If an athlete does not restore endogenous testosterone levels by using third-party products, to which the company belongs Clomiphene Iran Hormone, then later there are processes “rollback”, ie loss of physical strength and muscle size due to increased catabolism. This feature of the male body is due to the fact that the fall in testosterone levels, gaining a dominant position cortisol hormone that promotes catabolic metabolism. The process of losing weight gained and power performance is also called the “post-steroid disruption”.
Clomiphene Citrate: application
As already mentioned, the way out of this situation will be receiving anti-estrogen. Drink tablet is in the first weeks after the completion of steroids, as it is the time for an athlete it is important to compensate for the loss in the synthesis of natural testosterone, to minimize the consequences ( “rollback phenomenon”).
Clomiphene Citrate Dosages vary and depend primarily on the individual susceptibility of the athlete.Experienced athletes are recommended to use a dose of 50-100 mg of antiestrogen per day (needs), receiving course varies from 2 to 5 weeks, it is for such period will manifest the full effect of the tablets.
In addition to its indisputable beneficial effects on completion of a course of steroids, Iran Clomiphene Citrate can help the athlete to struggle with high levels of estrogen in the blood directly during treatment of steroid drugs. High levels of estrogen during this period can lead to serious consequences, including the development of gynecomastia. Clomiphene is due to their blocking action on estrogen (no effect on the flavoring process itself, ie the transformation of estrogens from androgens) copes with similar symptoms. So great to prevent estrogen-side effects.
Clomiphene Citrate is typically prescribed for women to aid in ovulation. In men, the application of Clomid causes an elevation of follicle stimulating hormone and luteinizing hormone. As a result, natural testosterone production is also increased.
This effect is obviously beneficial to the athlete, especially at the conclusion of a steroid cycle when endogenous testosterone levels are subnormal. When an athlete discontinues the use of steroids, his testosterone levels will most likely be suppressed. If endogenous testosterone levels are not brought to normal, a dramatic loss in size and strength may occur. Clomid plays a crucial role in preventing this crash in athletic performance.
Bodybuilders find that a daily intake of 50-100 mg of clomiphene citrate over a two week period will bring endogenous testosterone production back to an acceptable level. Clomid will gradually raise testosterone levels over its period of intake. Since an immediate boost in testosterone is often desirable, athlete will commonly use HCG (human choronic gonadotropin) for a couple of weeks, and the continue treatment with Clomid.
Clomid is also effective as an anti-estrogen. Most athletes will suffer from an elevated estrogen level at the conclusion of a cycle. A high estrogen level combined with a low testosterone level puts an athlete in serious risk of developing gynocomastia. With the intake of Clomid, the athlete gets the dual effect of blocking out some of the effects of estrogen, while also increasing endogenous testosterone production.